「relations」の検索結果

Density, whitespace, rhythm, and how space communicates relationships between elements. Use when building layouts, evaluating spacing consistency, or diagnosing why a design feels cramped or empty.

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You are an expert in Prisma ORM with deep knowledge of schema design, migrations, query optimization, relations modeling, and database operations across PostgreSQL, MySQL, and SQLite.

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Expert guidance for designing, implementing, migrating, and debugging SwiftData persistence in Swift and SwiftUI apps. Use when working with @Model schemas, @Relationship/@Attribute rules, Query or FetchDescriptor data access, ModelContainer/ModelContext configuration, CloudKit sync, SchemaMigrationPlan/history APIs, ModelActor concurrency isolation, or Core Data to SwiftData adoption/coexistence.

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Implement, review, or improve data persistence using SwiftData. Use when defining @Model classes with @Attribute, @Relationship, @Transient, @Unique, or @Index; when querying with @Query, #Predicate, FetchDescriptor, or SortDescriptor; when configuring ModelContainer and ModelContext for SwiftUI or background work with @ModelActor; when planning schema migrations with VersionedSchema and SchemaMigrationPlan; when setting up CloudKit sync with ModelConfiguration; or when coexisting with or migrating from Core Data.

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Design and generate Convex database schemas with proper validation, indexes, and relationships. Use when creating schema.ts or modifying table definitions.

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Prisma ORM expert for schema design, migrations, query optimization, relations modeling, and database operations. Use PROACTIVELY for Prisma schema issues, migration problems, query performance, relation design, or database connection issues.

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Expert C4 Component-level documentation specialist. Synthesizes C4 Code-level documentation into Component-level architecture, defining component boundaries, interfaces, and relationships.

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Perform comprehensive cloud asset inventory and relationship mapping using Cartography to build a Neo4j security graph of infrastructure assets, IAM permissions, and attack paths across AWS, GCP, and Azure.

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Busabase-backed App-in-Skill CRM operator for contacts, companies, deals, and agent-drafted follow-ups in a reviewable follow-up queue. Use when the user invokes $kelly-crm or /kelly-crm, mentions CRM, pipeline, contacts, companies, deals, follow-ups, relationship management, meeting-note capture, outreach drafts, next steps, or wants to review/approve agent-drafted follow-up messages before they are sent through other channels.

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Use when asked to map, crosswalk, align, compare, or gap-analyze any two cybersecurity frameworks, control catalogs, or regulatory requirements using NIST IR 8477 Set-Theory Relationship Mapping (STRM). Triggers on terms like "map controls", "crosswalk", "framework alignment", "gap analysis", or producing a STRM CSV output file.

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Detect abuse of legitimate Windows binaries (LOLBins) used for living off the land attacks. Monitors process creation, command-line arguments, and parent-child relationships to identify suspicious LOLBin execution patterns.

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General mathematical methods skill for symbolic and numerical work; use when solving equations or deriving relationships.

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General Signals scout for PostHog projects. Cross-product explorer that scans a team's project and emits findings into the Signals inbox. Sibling signals-scout-* specialists each watch a single product surface in depth; this scout looks for cross-product correlations and explores the surfaces no specialist covers. Each scout runs on its own schedule (default hourly), so general fires independently of the specialists over time.

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Design database schemas — tables, relationships, indexes, constraints, and ORM setup. Covers relational design, normalization, and common patterns.

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KEGG-based disease-drug-variant network research. Connects diseases to causal genes, drugs to molecular targets, and variants to pathways using KEGG's editorially curated databases (KEGG Disease, Drug, Network, Variant, Pathway). Use for drug repurposing via shared pathways, mechanistic disease-gene-drug networks, and pathway-based target discovery. Distinguishes direct (binding) vs indirect (pathway co-membership) drug-target relationships.

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Infer gene regulatory networks (GRNs) from gene expression data using scalable algorithms (GRNBoost2, GENIE3). Use when analyzing transcriptomics data (bulk RNA-seq, single-cell RNA-seq) to identify transcription factor-target gene relationships and regulatory interactions. Supports distributed computation for large-scale datasets.

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Biological knowledge: query curated assertions and reference databases for genes, variants, pathways, regulation, cell types, reference atlases, perturbation-derived relationships, phenotypes, therapeutic targets, drugs, interactions, annotations, signatures, compounds, and predicted structures. Use for identifiers, marker sets, gene sets, associations, ontologies, database facts, and cross-database coverage.

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Authoring and managing Agentforce agent definitions using the declarative Agent Script DSL (.agent files) and associated metadata types. Use when creating agents in source control, debugging agent metadata, or understanding the metadata lifecycle of GenAiPlugin/GenAiPlanner/BotVersion types. Triggers: 'how do I deploy an Agentforce agent using source control', 'what metadata types make up an Agentforce agent', 'agent test run command failing in CI pipeline', 'GenAiPlugin vs GenAiPlanner metadata relationship'. NOT for Apex-based agent actions (use custom-agent-actions-apex). NOT for UI-based agent creation (use agentforce-agent-creation).

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Compare gene co-expression and regulatory networks between biological conditions to find rewired relationships using DiffCorr, DiffCoEx, DINGO/iDINGO, and CoDiNA. Covers the differential-connectivity-is-not-differential-expression distinction, the pairwise multiple-testing explosion, marginal vs partial (direct) rewiring, and the underpowered-rewiring failure mode. Use when comparing co-expression networks between disease vs control, treatment, or developmental stages, or finding hub genes that rewire without changing mean expression. For single-condition modules see coexpression-networks; for differential expression of means see differential-expression/de-results.

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Analyzes species-environment relationships with constrained ordination (CCA, RDA, db-RDA), variance partitioning, indicator species (indicspecies IndVal.g group-equalized), PERMANOVA paired MANDATORILY with PERMDISP (Anderson & Walsh 2013; dispersion confounds centroid tests), Joint Species Distribution Models (HMSC, sjSDM, gjam) with explicit rejection of "residual covariance equals biotic interaction", phylogenetic community ecology (SES_MPD/MNTD), trait-environment via RLQ + fourth-corner with corrected modeltype=6 (Dray 2014), bipartite network metrics (NODF, modularity) with curveball null (Strona 2014), and Mantel-test replacements (dbRDA, GDM) for spatial data. Use when testing how environmental gradients structure communities, identifying habitat indicator taxa, partitioning variance among predictors, deciding whether PERMANOVA significance is location vs dispersion, picking among HMSC/sjSDM/gjam, or replacing Mantel tests for landscape data.

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Batch effect correction for CRISPR screens covering ComBat empirical-Bayes, RUV, SVA, control-sgRNA normalization, and the model-based alternative of including batch as a covariate in MAGeCK MLE or Chronos. Covers screen-specific batch sources (passage cohort, library lot, infection day, sequencing run, Cas9 lot, FBS lot), PCA + variance-decomposition diagnostic to decide if correction is needed, when correction harms biology by over-correcting condition into batch, limma removeBatchEffect for visualization-only correction, and relationship to multi-condition design matrices. Use when combining screens for joint analysis, when passage cohort confounds biology, when DepMap-style panels need Chronos with batch covariates, when picking ComBat vs RUV, or when correction harms biology and should be replaced with explicit covariate modeling.

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Find cross-database references between NCBI databases using Biopython Bio.Entrez (ELink). Use when navigating gene to protein/structure, sequence to publication, PubMed to GEO, BioProject to SRA runs, or discovering all link relationships for a record. Covers linkname semantics, cmd= variants, asymmetric link warnings, neighbor_history for >200 input IDs, and per-database link tables.

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Designs and analyzes in vivo CRISPR screens in animal tumor models, organoids, and immune-cell adoptive transfers. Covers bottleneck math (250x cells/sgRNA requires ~25M cells implanted; impossible for most syngeneic models, forcing focused libraries), focused library design (Manguso 2017 Nature 547:413 immune screen; Chen 2015 tumor screens), CRISPR-StAR intrinsic-control screening (Uijttewaal 2025 Nat Biotechnol 43:1848), clonal-dynamics-limited detection, tumor-explant DNA recovery, syngeneic vs xenograft vs PDX considerations, and the relationship to downstream MAGeCK / drugZ analysis. Use when designing in vivo CRISPR screens for tumor / immune / metastasis biology, choosing focused vs genome-wide for animal models, addressing bottleneck-induced clonal collapse, picking the syngeneic / xenograft / PDX model, integrating in vivo with in vitro results, or applying CRISPR-StAR for animal experiments.

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Designs and analyzes combinatorial CRISPR screens covering paired-Cas9 (Big Papi, Najm 2018), enhanced AsCas12a multiplex (enCas12a, DeWeirdt 2021), in4mer 4-guide-array Cas12a (Esmaeili Anvar N et al 2024 Nat Commun 15:3577) and the Inzolia paralog-pair library, paralog-buffering detection (Dede 2020 Genome Biol; Thompson 2021 Cell Reports 36:109597), genetic-interaction (GI) scoring as observed_double_LFC minus expected_additive_double_LFC, synthetic-lethal and synthetic-rescue interaction interpretation, the half-of-essentiality buffered by paralogs phenomenon, multiplex screen statistical analysis with MAGeCK MLE interaction terms, and the relationship to single-cell combinatorial Perturb-seq. Use when designing a paralog or pathway-pair screen, choosing between paired-Cas9 (Big Papi) and Cas12a multiplex (Inzolia), interpreting genetic interaction scores, identifying synthetic-lethal targets for drug development, or scaling beyond single-gene CRISPR screens.

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